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¿Qué debe preguntar a su médico sobre la leucemia linfocítica aguda? Es importante sostener diálogos sinceros y honestos con su médico. Existen cuatro tipos principales de leucemia: Leucemia linfoblástica (linfocítica) aguda (ALL, por sus siglas en inglés). Leucemia mieloide (mielógena) aguda. Sin embargo, el aspecto cumbre de su estudio fue el descubrimiento, hace 30 años, de que la variedad más común en el niño, la leucemia linfocítica aguda.

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Blood, 81pp. The role of cytogenetics in this molecular era. Abnormalities of the long arm of chromosome 6 in childhood acute lymphoblastic leukemia.

¿Qué avances hay en la investigación y el tratamiento de la leucemia linfocítica aguda?

This study focuses on the most important chromosomal abnormalities found in childhood ALL and their prognostic qguda therapeutic implications. Nonrandom abnormalities of chromosome 9p in childhood acute lymphoblastic leukemia: Centric and pericentric chromosome rearrangements in hematopoietic malignancies. Blood, 65pp. Immunologic, cytogenetic, and clinical characterization of childhood acute lymphoblastic leukemia with the t 1;9 q23;p13 or its derivative.

Br J Haematol, 90pp. Si continua navegando, consideramos que acepta su uso.

Preguntas que deben formularse acerca de la leucemia linfocítica aguda

Cytogenetic analysis of blast cells in childhood acute lymphoblastic leukemia has led to the recognition of specific non-random chromosomal abnormalities with prognostic value. High survival rate in advanced-stage-B-cell lymphomas and leukemias without CNS involvement with a short intensive polychemotherapy: Blood, 76pp.


Are you a health professional able to prescribe or dispense drugs? Cytogenetic abnormalities in acute lymphoblastic leukemia. Chromosomal localization of human leukocyte, fibroblast, and immune interferon genes by means of in situ hybridization.

Blood, 74pp. Current status of cytogenetic research in childhood acute lymphoblastic leukemia. Blood, 87pp. Chromosomal translocations involving the E2A gene in acute lymphoblastic leukemia: Hyperdiploid acute lymphoblastic leukemia in children.

Clinical and cytogenetic characteristics and treatment outcome: Identification of a gene, MLL, that spans the breakpoint in 11q23 translocations associated with human leukemias. Trisomy of leukemic cell chromosomes 4 and 10 identifies children with B-progenitor cell linvocitica lymphoblastic leukemia with a very low risk of treatment failure. Genomics, 17pp.

Oncogene, 7pp. You can change the settings or obtain more information by clicking here.

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Translocation t 9;22 is associated with extremely poor prognosis in intensively treated children with acute lymphoblastic leukemia. Poor prognosis of children with pre-B acute lymphoblastic leukemia is associated with the t 1;19 q23,p Blood, 84pp.


Nonrandom involvement of the 12p12 breakpoint in chromosome abnormalities of childhood acute lymphoblastic leukemia. Translocation 12;22 p13;q11 in myeloproliferative disorders results in fusion of the ETS-like Tel gene on 12p13 to the MN1 gene on 22q Fusion with EA2 converts the Pbx1 homeodomain protein into a constitutive transcriptional activator in human leukemias carrying the 1; Sangre, 44pp. Cytogenetic abnormalities in childhood acute lymphoblastic leukemia correlates with clinical features and treatmentoutcome.

Br J Haematol,pp. Leukemia, 7pp. Most patients with ALL show karyotype abnormalities, either in chromosome number ploidy or as structural changes such as translocations, inversions, or deletions. J Clin Oncol, 9pp.

J Clin Kinfocitica, 12pp. This has led to the administration of alternative therapies according to risk. Unifirm approch to leucmeia classification and treatment assignment to children with acute lymphoblastic leukemia. Blood, 75pp.

Collaborative study of karyotypes in childhood acute lymphoblastic leukemia. Certain karyotypes are associated with a favorable prognosis while others indicate a poor outcome. Br Med J, 2 agudx, pp.